DISK HEMORRHAGES MAY NOT INDICATE INADEQUATE IOP CONTROL

This was an evaluation of patients within the Early Manifest Glaucoma Trial. It's purpose was:

"To evaluate the effect of intraocular pressure (IOP)-reducing treatment on the development of disc hemorrhages in patients with glaucoma."

The conclusion was:

"IOP-reducing treatment was unrelated to the presence or frequency of disc hemorrhages. The results may suggest that disc hemorrhages cannot be considered an indication of insufficient IOP-lowering treatment, and that glaucoma progression in eyes with disc hemorrhages cannot be totally halted by IOP reduction. The results also suggest that disc hemorrhages do not occur in all patients with glaucoma."

CALCIUM ANTAGONIST MAY HELP IN LOW TENSION GLAUCOMA

According to this study, a calcium antagonist, nilvadipine, taken orally, may decrease the rate of mean deviation depression over time in patients with glaucoma.
Further studies are needed for confirmation and to determine if benefits outweigh risks.
Theoretically calcium antagonists would increase bloodflow of optic nerve vasculature.

INTENSE INITIAL TREATMENT OF DIABETES MAY CAUSE TRANSIENT HYPEROPIA

Transient Hyperopia after Intensive Treatment of Hyperglycemia in Newly Diagnosed Diabetes

This interesting paper shows that there is a hyperopic shift with aggressive initial treatment of diabetes, but that this shift apparently disappears between two to three months.

NEVANAC IMPROVES CSME

Journal of Clinical Ophthalmology - Dove Medical Press:

The results from this small study suggests that nepafenac 0.1% may have activity against diabetic macular edema in a significant way.